Himalaya Turmeric Haridra / Curcumin / Haldi (Curcuma longa)
Benefits of Turmeric: detoxifier, anti-oxidant, anti-inflammatory, anti-bacterial, anti-septic. Turmeric is also considered very beneficial in preventing Alzheimers, cancer and liver problems.
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Himalaya Turmeric is a pure herb extract.
Turmeric or curcumin (Latin name: Curcumin longa) is a herb that is used extensively in south asia as a food ingredient as well as a medicine.
Studies indicate that the use of turmeric in Indian dishes is the likely reason that India has the lowest incidence of Alzheimer's disease in the world where only 1% of those over 65 are affected by it.
Research has shown that turmeric helps to ward off Alzheimer's disease by reducing buildup of amyloid protein or plaque in the brain as well as reducing inflammation in neurologic tissue.
Scientists also consider turmeric to be the premier anti-cancer herb.
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Himalaya Turmeric 60 Capsules per Bottle ; 250mg per capsule (Pure Herb Extract)
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There are over 1300 published studies documenting turmeric's medicinal properties including its benefits against cancer. Read below for new research on curcumin
Benefits of Turmeric
- Turmeric is a powerful anti-oxidant and prevents cell aging. It is 300 times more potent than Vitamin E
- Turmeric stimulates the immune system
- Turmeric is a potent natural anti-inflammatory that helps in the treatment of arthritis and rheumatoid arthritis.
- Turmeric shows great promise in preventing several types of cancer including breast cancer, leukemia, prostate cancer and melanoma
- Turmeric is excellent for skin health and treatment of skin disorders such as psoriasis
- Turmeric is a natural anti-septic and anti-bacterial agent that helps in treating cuts, infections and burns when applied topically
- Turmeric has been shown to prevent Alzheimers by reducing plaque in the brain
- Turmeric is a detoxifier and helps in liver disorders
- Turmeric promotes weight loss by metabolizing fat.
- Turmeric is said to boost the effectiveness of chemotherapy especially the chemo drug paclitaxel and reduces its side effects.
Turmeric in Ayurveda:
According to Ayurveda, turmeric's gunas or qualities are that it is laghu (dry) and ruksha (rough). Its rasas (taste) is katu (pungent) and tikta (bitter). It has ushna virya and is tridoshic at normal dosages.
Turmeric principally treats the skin, heart, liver and lungs. It was recommended by Sushruta epilepsy and bleeding disorders.
Charaka recommended it for skin diseases, to purify the bodymind, and to help
the lungs expel Kapha.
Turmeric Facts:
Botanical Name : : CURCUMA LONGA
Family Name: : ZINGIBERACEAE
Common Name: : TURMERIC, CURCUMIN, HARIDRA, HALDI
Turmeric is a rhizome or root belonging to the ginger family and grows in South Asia. The roots can be eaten raw or ground to a powder after drying. The final powder is brilliant yellow in color and used as a spice in flavoring foods and imparting the yellow color.
Turmeric is also used as a dye and in cosmetics. It has a part in many religious ceremonies in India. It has many medicinal properties and is used as a household anti-septic and anti-inflammatory medicine.
Turmeric's properties include being an alterative, analgesic, antibacterial, anti-inflammatory, antitumor,
anti-allergic, antioxidant, antiseptic, antispasmodic, appetizer, astringent, cardiovascular, carminative,
cholagogue, digestive, diuretic, stimulant, and vulnerary.
Directions for taking Himalaya Turmeric
Take 1 or 2 capsules twice daily with meals. Allow several weeks for benefits. The use of natural products provides progressive but long-lasting results.
Pure Turmeric from Himalaya Herbals
Himalaya Turmeric is from the renowned Himalaya Herbals brand endorsed by over 250,000 doctors worldwide and used by customers in over 60 countries. Himalaya Herbals products have been researched clinically and standardized to guarantee bioequivalence. Bioequivalence refers to ensuring that the product on the market is equivalent to the one on which clinical trials were successfully conducted. Himalaya Herbal Healthcare uses chromatographic fingerprinting, one of the most sophisticated standardization techniques, to ensure consistent quality and performance
Turmeric Research and Clinical Studies
NEW Research now shows how curcumin works
Using solid-state NMR spectroscopy researchers at the University of Michigan revealed that molecules of turmeric insert themselves into cell membranes causing them to become more stable and better able to resist infection from microbes.
According to the study published in the Journal of the Americal Chemical Society "Curcumin is the active ingredient of turmeric powder, a natural spice used for generations in traditional medicines. Curcumin’s broad spectrum of antioxidant, anticarcinogenic, antimutagenic, and anti-inflammatory properties makes it particularly interesting for the development of pharmaceutical compounds. Because of curcumin’s various effects on the function of numerous unrelated membrane proteins, it has been suggested that it affects the properties of the bilayer itself. A combination of solid-state NMR and differential scanning calorimetry experiments shows curcumin has a strong effect on membrane structure at low concentrations. Curcumin inserts deep into the membrane in a transbilayer orientation, anchored by hydrogen bonding to the phosphate group of lipids ... and forms higher order oligomeric structures in the membrane that span and likely thin the bilayer. Curcumin promotes the formation of the highly curved inverted hexagonal phase, which may influence exocytotic and membrane fusion processes within the cell"
The Curry Spice Curcumin Reduces Oxidative Damage and Amyloid Pathology in an Alzheimer Transgenic Mouse.
Departments of Medicine and Neurology, University of California, Los Angeles, Los Angeles, California 90095, and Greater Los Angeles Veterans Affairs Healthcare System, Geriatric Research, Education and Clinical Center, Sepulveda, California 91343.
Inflammation in Alzheimer's disease (AD) patients is characterized by increased cytokines and activated microglia. Epidemiological studies suggest reduced AD risk associates with long-term use of nonsteroidal anti-inflammatory drugs (NSAIDs). Whereas chronic ibuprofen suppressed inflammation and plaque-related pathology in an Alzheimer transgenic APPSw mouse model (Tg2576), excessive use of NSAIDs targeting cyclooxygenase I can cause gastrointestinal, liver, and renal toxicity. One alternative NSAID is curcumin, derived from the curry spice turmeric. Curcumin has an extensive history as a food additive and herbal medicine in India and is also a potent polyphenolic antioxidant. To evaluate whether it could affect Alzheimer-like pathology in the APPSw mice, we tested a low (160 ppm) and a high dose of dietary curcumin (5000 ppm) on inflammation, oxidative damage, and plaque pathology. Low and high doses of curcumin significantly lowered oxidized proteins and interleukin-1beta, a proinflammatory cytokine elevated in the brains of these mice. With low-dose but not high-dose curcumin treatment, the astrocytic marker GFAP was reduced, and insoluble beta-amyloid (Abeta), soluble Abeta, and plaque burden were significantly decreased by 43-50%. However, levels of amyloid precursor (APP) in the membrane fraction were not reduced. Microgliosis was also suppressed in neuronal layers but not adjacent to plaques. In view of its efficacy and apparent low toxicity, this Indian spice component shows promise for the prevention of Alzheimer's disease.
Developing curcumin into a viable therapeutic for lymphoma.
Expert Opin Investig Drugs. 2009 Jan;18(1):57-67.
Emerging evidence suggests that natural plant ingredients have played an important role in the healthcare of many countries. Several of these natural plant products possess therapeutic potential for various diseases including cancer. Curcumin is the pigment of turmeric, a well-known chemopreventive agent that has been shown to suppress the proliferation of a wide variety of tumor cells, including lymphoma. Curcumin has been shown to have cancer chemopreventive potential against a variety of tumors via targeting key survival pathways that are aberrantly activated in cancer cells. METHODS: This review discusses therapeutic potential of curcumin in malignancies of lymphoma as well as therapeutic implications of the recent advances in the field. RESULTS/CONCLUSION: Dietary-compound curcumin hardwires to multiple cellular processes. Suppression of cell proliferation, induction of apoptosis, and inhibition of metastasis are considered to be the major mechanisms underlying its anticancer properties
Antiproliferative property and apoptotic effect of xanthorrhizol on MDA-MB-231 breast cancer cells.
Anticancer Res. 2008 Nov-Dec;28(6A):3677-89.
Xanthorrhizol is a natural sesquiterpenoid compound isolated from the rhizome of Curcuma xanthorrhizza Roxb (Zingerberaceae). Recent studies of xanthorrhizol in cell cultures strongly support the role of xanthorrhizol as an antiproliferative agent. In our study, we tested the antiproliferative effect of xanthorrhizol using different breast cancer cell lines. The invasive breast cancer cell line, MDA-MB-231, was then selected for further investigations. Treatment with xanthorrhizol caused 50% growth inhibition on MDA-MB-231 cells at 8.67 +/- 0.79 microg/ml as determined by sulforhodamine B (SRB) assay. Hoechst 33258 nuclear staining assay showed the rate of apoptosis of MDA-MB-231 cells to increase in response to xanthorrhizol treatment. Immunofluorescence staining using antibody MitoCapture and fluorescein isothiocyanate (FITC)-labeled cytochrome c revealed the possibility of altered mitochondrial transmembrane potential and the release of cytochrome c respectively. This was further confirmed by Western-blotting, where cytochrome c was showed to migrate from mitochondrial fraction to the cytosol fraction of treated MDA-MB-231 cells. Caspase activity assay showed the involvement of caspase-3 and caspase-9, but not caspase-6 or caspase-8 in MDA-MB-231 apoptotic cell death. Subsequently, cleavage of PARP-1 protein is suggested. These data suggest treatment with xanthorrhizol modulates MDA-MB-231 cell apoptosis through the mitochondria-mediated pathway subsequent to the disruption of mitochondrial transmembrane potential, release of cytochrome c, activation of caspase-3 and caspase-9, and the modulation of PARP-1 protein.
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